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Relationship of Acidosis to Other Feedlot Ailments^{1 ,2,}

Kansas State University, Manhattan 66506

Abstract

Laminitis often follows lactic acidosis and is accompanied by increased blood and rumen histamine. However, since histamine is poorly absorbed and absorbed histamine is rapidly metabolized, and since high levels of oral histamine have not produced laminitis, it is unlikely that ruminal histamine causes laminitis.

Lactic acidosis leads to rumenitis, which, in turn, leads to liver abscesses because the causative microorganism can now cross the rumen wall into portal circulation. In cattle, hair ingested during grooming may penetrate the rumen wall and aid in this passage. Immunizing cattle against liver abscesses may be possible.

Polioencephalomalacia occurs because an enzyme, thiaminase, develops in the rumen, catalyzing the production of a thiamin antagonist. Lactic acidosis may set up ruminal conditions that encourage this chain of events.

¹ Contribution No. 501, Department of Animal Science and Industry, Kansas Agricultural Experiment Station, Manhattan 66506.

² Presented at the Symposium on Acidosis in Feedlot Cattle at the 67th Annual Meeting of die American Society of Animal Science, Fort Collins, July 28 to 30, 1975. Co-sponsored by die American Society of Animal Science and the Committee on Animal Nutrition, National Research Council.

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